Conclusion

Infection plays a role in carcinogenesis via mutation and modification of gene expression through inflammation. Helicobacter pylori infection in the stomach is prevalent in the world and constitutes a major risk factor for gastric carcinogenesis. Helicobacter pylori cause acute gastritis and then chronic gastritis, with associated mucosal atrophy and intestinal metaplasia. Approximately 10% of the people with H. pylori infection are reported to suffer from gastric adenocarcinoma eventually. Helicobacter pylori is a promoter in carcinogenesis, but it is not established yet whether it is an initiator. The International Agency for Research on Cancer declared H. pylori as a definite carcinogen based on the epidemiologic studies. Chemopreventive intervention as well as eradication of H. pylori is necessary to decrease the incidence of gastric cancer.

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